How Does Cannabis Consumption Affect Insomnia?

There’s a reason more insomnia sufferers are turning to cannabis. You toss and turn, count sheep, and negotiate (“If I can fall asleep by 3 a.m., I’ll at least get four hours of sleep”). Ten minutes becomes a half hour. A half hour becomes an hour. Before you know it, the sun is coming up. Insomnia is its own unique agony, but now that the stigma of cannabis is slowly lifting, more people are seeking out its therapeutic sedating properties.
Acute insomnia — which usually only lasts a night or two — happens to nearly all of us. It’s usually triggered by a stressful external event. But, chronic insomnia — regularly having three or more restless nights per week over the course of the month — affects a billion people worldwide.

Interestingly, insomnia affects women at twice the rate as men. And, it doesn’t get better with age. Half of all seniors are regularly affected by insomnia. Beyond feeling sleepy and irritable the next day, chronic insomnia is associated with some serious long-term health issues: cardiovascular disease, diabetes, and obesity.

So what causes insomnia, what are the traditional treatment options, and in what ways might cannabis be a healthier, viable alternative?

Causes of Insomnia:

Insomniacs can have either primary or secondary insomnia.

Primary insomnia is a standalone condition — it wasn’t caused by another health condition.
Secondary insomnia is associated with a secondary health condition or substance (for example, depression, pain, alcohol, or a prescription medication).
The number one cause of insomnia is stress. While we can’t control every stressful external factor in our lives, there are healthy ways to deal with the stress. Dr. Rachna Patel, a physician from Walnut Creek, California, who has personally dealt with years of insomnia, notes, “Anything you can do to reduce stress will also help you sleep better. Get out for a jog. Swim. Eat better. Do relaxation exercises or meditate. Even if you still need a sleep aid like cannabis, lifestyle changes will improve your overall health!”

Traditional Treatment Options for Insomnia:
Dr. Patel suggests, “Before trying medication, consider making lifestyle changes including setting a regular sleep schedule, getting more exercise, [and] eating healthier.” Nonetheless, Patel has observed that “some patients have so much difficulty sleeping that they just need a medication to help them.”

Prescription medications such as Zolpidem (Ambien) and Zaleplon (Sonata) have grown in popularity over the years, but they may not be that effective. One study by the National Institutes of Health found that sleeping pills, on average, only add 11 minutes of sleep time and shorten the time it takes to fall asleep by a mere 13 minutes.

Worse, they can come with serious adverse side effects and health consequences. According to the Department of Health and Human Services, over the last two decades there’s been a dramatic increase in prescription sleep aid-related emergency room visits.

Likewise, benzodiazepines such as Xanax and Valium, which are approved for sleep, are highly addictive and potentially dangerous. According to the Centers for Disease Control and Prevention (CDC), in 2013 benzos were involved in 30% of lethal drug overdoses, second only to opioids.

Some also claim that natural supplements, such as melatonin, valerian root, lemon balm, or chamomile are helpful in falling asleep.

Can Cannabis Treat Insomnia?

Dr. Patel turned to cannabis after being prescribed Ambien. Afraid of the potential side effects, she found research validates what cannabis users have long suspected: cannabis helps people sleep.

Strangely, the National Institute on Drug Abuse (NIDA), National Institutes of Health (NIH), and Big Pharma’s Sanofi-Aventis may agree. They funded a study that showed consuming THC enabled subjects to fall asleep easier and more quickly.

Here’s more of the evidence:

Easier time falling asleep. As far back as 1973, research has documented subjects falling asleep quicker after ingesting THC. More recently, a 2013 study of healthy subjects validated earlier findings.
Sleep longer and better. Early studies have demonstrated the efficacy of cannabinoids in aiding sleep. One study of THC found that subjects experienced fewer interruptions over the night and some decrease in awakenings during the first half of the night.
Enjoy deeper sleep. Cannabis can positively impact the sleep cycle. Studies prove THC can increase deep sleep. Why is this important? Because scientists believe deep sleep plays a vital role in our body’s natural restoration process.
Better breathing while sleeping. Roughly 17% of men and 9% of women regularly have breathing problems when they sleep – a condition called sleep apnea – and most are never diagnosed. However, early research published in January 2013 by Frontiers in Psychology shows cannabis may help people breathe easier when they sleep. Who knows? Maybe someday sleep apnea sufferers can swap out their CPAP mask for a THC-infused brownie (but don’t count on Medicare to cover the cost yet).

How Does Cannabidiol (CBD) Affect Sleep?
vidence of cannabidiol, or CBD, as a sleep aid has been contradictory. In one study, CBD – which is non-psychoactive – seemed to be effective as a “wake-inducing agent,” meaning it can make you feel more alert, the opposite effect of what an insomniac wants.

However, others who participated in the study reported that ingesting CBD-rich extracts or tinctures a few hours prior to bed had a relaxing effect that allowed them to sleep better at night. According to Project CBD, some patients with sleep issues report that “ingesting a CBD-rich tincture or extract a few hours before bedtime has a balancing effect that facilitates a good night’s sleep.”

The key is finding the right strain, blend, product, and dose for you. Everyone’s body responds to cannabis differently, so it may take a little trial-and-error before finding the perfect fit. Try a heavy indica or an edible. Consider something with a little CBD. See what happens when you dose a little instead of a lot. You might be surprised to find that your ultimate sleep remedy isn’t what you thought it would be.

Source : Leafly

3 Studies That Show Cannabis Grows Brain Cells

Neuropsychopharmacology
July 2013
The anxiolytic effect of cannabidiol on chronically stressed mice depends on hippocampal neurogenesis: involvement of the endocannabinoid system
Abstract
Cannabidiol (CBD), the main non-psychotomimetic component of the plant Cannabis sativa, exerts therapeutically promising effects on human mental health such as inhibition of psychosis, anxiety and depression.
However, the mechanistic bases of CBD action are unclear.
Here we investigate the potential involvement of hippocampal neurogenesis in the anxiolytic effect of CBD in mice subjected to 14 d chronic unpredictable stress (CUS).
Repeated administration of CBD (30 mg/kg i.p., 2 h after each daily stressor) increased hippocampal progenitor proliferation and neurogenesis in wild-type mice. …
CBD administration prevented the anxiogenic effect of CUS in wild type but not in GFAP-TK mice as evidenced in the novelty suppressed feeding test and the elevated plus maze.
This anxiolytic effect of CBD involved the participation of the CB1 cannabinoid receptor, as CBD administration increased hippocampal anandamide levels and administration of the CB1–selective antagonist AM251 prevented CBD actions.
Studies conducted with hippocampal progenitor cells in culture showed that CBD promotes progenitor proliferation and cell cycle progression and mimics the proliferative effect of CB1 and CB2 cannabinoid receptor activation. …
These findings support that the anxiolytic effect of chronic CBD administration in stressed mice depends on its proneurogenic action in the adult hippocampus by facilitating endocannabinoid-mediated signalling.
http://journals.cambridge.org/action/displayAbstract?fromPage=online&aid=8930251

PLOS ONE (Public Library of Science)
May 2013
Activation of Type 1 Cannabinoid Receptor (CB1R) Promotes Neurogenesis in Murine Subventricular Zone Cell Cultures
Abstract
The endocannabinoid system has been implicated in the modulation of adult neurogenesis.
Here, we describe the effect of type 1 cannabinoid receptor (CB1R) activation on self-renewal, proliferation and neuronal differentiation in mouse neonatal subventricular zone (SVZ) stem/progenitor cell cultures.
Expression of CB1R was detected in SVZ-derived immature cells (Nestin-positive), neurons and astrocytes.
Stimulation of the CB1R … increased self-renewal of SVZ cells, as assessed by counting the number of secondary neurospheres … Moreover, … treatment for 48 h, increased proliferation …
Surprisingly, stimulation of CB1R … also promoted neuronal differentiation (without affecting glial differentiation), at 7 days, as shown by counting the number of NeuN-positive neurons in the cultures.
Moreover, by … a method that allows the functional evaluation of neuronal differentiation, we observed an increase in neuronal-like cells.
This proneurogenic effect was blocked when SVZ cells were co-incubated with … the CB1R antagonist AM 251, for 7 days, thus indicating that this effect involves CB1R activation.
In accordance with an effect on neuronal differentiation and maturation … also increased neurite growth …
Taken together, these results demonstrate that CB1R activation induces proliferation, self-renewal and neuronal differentiation from mouse neonatal SVZ cell cultures.
http://www.plosone.org/article/info%3Adoi/10.1371/journal.pone.0063529

The Journal of Clinical Investigation
November 2005
Cannabinoids promote embryonic and adult hippocampus neurogenesis and produce anxiolytic- and antidepressant-like effects
Abstract
The hippocampal dentate gyrus in the adult mammalian brain contains neural stem/progenitor cells (NS/PCs) capable of generating new neurons, i.e., neurogenesis.
Most drugs of abuse examined to date decrease adult hippocampal neurogenesis, but the effects of cannabis (marijuana or cannabinoids) on hippocampal neurogenesis remain unknown.
This study aimed at investigating the potential regulatory capacity of the potent synthetic cannabinoid HU210 on hippocampal neurogenesis and its possible correlation with behavioral change.
We show that both embryonic and adult rat hippocampal NS/PCs are immunoreactive for CB1 cannabinoid receptors, indicating that cannabinoids could act on CB1 receptors to regulate neurogenesis.
This hypothesis is supported by further findings that HU210 promotes proliferation, but not differentiation, of cultured embryonic hippocampal NS/PCs likely via a sequential activation of CB1 receptors …
Chronic, but not acute, HU210 treatment promoted neurogenesis in the hippocampal dentate gyrus of adult rats and exerted anxiolytic- and antidepressant-like effects.
… suggesting that chronic HU210 treatment produces anxiolytic- and antidepressant-like effects likely via promotion of hippocampal neurogenesis.

Introduction
Cannabis (marijuana, hashish, or cannabinoids) has been used for medical and recreational purposes for many centuries and is likely the only medicine or illicit drug that has constantly evoked tremendous interest or controversy within both the public domain and medical research.
Cannabinoids appear to be able to modulate pain, nausea, vomiting, epilepsy, ischemic stroke, cerebral trauma, multiple sclerosis, tumors, and other disorders in humans and/or animals.
However, marijuana has been the most commonly used illicit drug in developed countries, producing acute memory impairment and dependence/withdrawal symptoms in chronic users and animal models.
Cannabis acts on 2 types of cannabinoid receptors, the CB1 and CB2 receptors, which are distributed mainly in the brain and immune system, respectively.
In the brain, CB1 receptors are also targeted by endogenous cannabinoids (i.e., endocannabinoids) such as anandamide (AEA), 2-arachidonylglycerol, and arachidonylethanolamide.

The recent discovery that the hippocampus is able to generate new neurons (i.e., neurogenesis) throughout the lifespan of mammals, including humans, has changed the way we think about the mechanisms of psychiatric disorders and drug addiction.

The subgranular zone of the dentate gyrus (SGZ) in the adult brain contains neural stem/progenitor cells (NS/PCs) capable of producing thousands of new granule cells per day.
We, and others, have shown that these newborn hippocampal neurons are functionally integrated into the existing neuroanatomical circuitry and are positively correlated with hippocampus-dependent learning and memory processes and the developmental mechanisms of stress and mood disorders. …

Chronic administration of the major drugs of abuse including opiates, alcohol, nicotine, and cocaine has been reported to suppress hippocampal neurogenesis in adult rats, suggesting a potential role of hippocampal neurogenesis in the initiation, maintenance, and treatment of drug addiction.
The recent finding of prominently decreased hippocampal neurogenesis in CB1-knockout mice suggests that CB1 receptor activation by endogenous, plant-derived, or synthetic cannabinoids may promote hippocampal neurogenesis.

However, endogenous cannabinoids have been reported to inhibit adult hippocampal neurogenesis.
Nevertheless, it is possible that exo- and endocannabinoids could differentially regulate hippocampal neurogenesis, as exo- and endocannabinoids act as full or partial agonists on CB1 receptors, respectively.

The goal of the present study was to test the hypothesis that the potent synthetic cannabinoid HU210 is able to promote hippocampal neurogenesis, leading to the anxiolytic and antidepressant effects of cannabinoids.
We demonstrate here that both HU210 and the endocannabinoid AEA promote proliferation of embryonic hippocampal NS/PCs without significant effects on their differentiation, resulting in more newborn neurons.
The effects of HU210 on adult hippocampal neurogenesis were quantified in freely moving rats and were correlated with behavioral testing.

We show that 1 month after chronic HU210 treatment, rats display increased newborn neurons in the hippocampal dentate gyrus and significantly reduced measures of anxiety- and depression-like behavior.
Thus, cannabinoids appear to be the only illicit drug whose capacity to produce increased hippocampal newborn neurons is positively correlated with its anxiolytic- and antidepressant-like effects.
http://www.jci.org/articles/view/25509
Source: Real Farmacy

Hemp & Ginger Cake

Ingredients:
150g dark soft brown sugar
50g black treacle or molasses
3 medium eggs
125ml sunflower oil
125g chopped glacé ginger
175g grated sweet potato
150g plain flour
75g hemp flour
3 tsp ground ginger
3 tsp baking powder
For the icing
200g full-fat cream cheese, cold
100g unsalted butter, softened
1 tsp ground cinnamon, plus extra
125g sifted icing sugar

Method:

Line the base of two 18cm round layer cake tins with discs of nonstick baking paper, and heat the oven to 180C (160C fan-assisted)/350F/gas mark 4. Beat the sugar, treacle and eggs until smooth and light, then beat in the oil and stir in the glacé ginger and sweet potato. Sift in the flours, ground ginger and baking powder, and fold through evenly.

Spoon the mixture into the tins and bake for 25-30 minutes, until a skewer comes out clean. Leave to cool in the tins. Beat half the cream cheese with the butter, cinnamon and icing sugar until smooth, then lightly beat in the remaining cheese until thick. Layer and top the cake with icing, and sprinkle with more cinnamon to serve.

Enjoy

The Importance of Enhancing Your Endocannabinoid System

Cannabis extracts are proving to be remarkably effective against a wide range of diseases for thousands of people. Unfortunately, there is a subset of the population that responds negatively or not at all. Numerous factors influence an individual’s unique response to cannabis medicine, including genetics. Some people may never be able to benefit from cannabis due to rare genetic mutations. However, a major cause of poor experiences may be controllable – the health of the endocannabinoid system.

Cannabis and the Endocannabinoid System
Cannabis is effective because it works through the endocannabinoid system, the function of which is to maintain homeostasis. Given this role, it may often be the best place to target for treating disease, which fundamentally is a state of non-homeostasis. For cannabis to work most effectively, the endocannabinoid system needs to be healthy.
While concentrated cannabis can directly improve endocannabinoid signaling, they are not a cure-all. Other restorative and enhancement techniques must be utilized as well. If the endocannabinoid system can be enhanced before or alongside cannabis extract therapy, the healing results are almost certain to improve considerably.
Enhancing Your Endocannabinoid System
There are two primary ways to strengthen your endocannabinoid system. The first is by avoiding stimuli that deplete endocannabinoid resources. For example, one of the functions the system mediates is the reduction of inflammation. Therefore, eating inflammatory foods like refined grains, sugar, and trans fats can eventually be overwhelming. Stress also recruits endocannabinoid resources, so poor nutrition combined with stress can be especially damaging. Eating an anti-inflammatory diet rich in fruits, vegetables, nuts, seeds, and healthy fat sources like olive oil, fish, and eggs is very helpful, as is engaging in stress-reducing activities like meditation and yoga.
The second pathway to empowerment is consuming things that up-regulate endocannabinoids or cannabinoid receptors. For example, probiotics increase CB2 receptors in intestinal cells and may even potentially reduce pain by acting through those receptors. Exercising, including running and biking, increases endocannabinoid levels. Olive oil, in addition to being anti-inflammatory, may help fight colon cancer by increasing CB1 receptors on the cancer cells.

Source: Medical Jane

Study Shows THC May Limit Damage Caused By Heart Attack

A recent study conducted by the Felsenstein Medical Research Center in Israel offered some new evidence of the medical value of Cannabis. The study, published in the Journal of Biochemical Pharmacology, was conducted to determine what effect small amounts of tetrahydrocannabinol (THC)has on heart protection. In order to do so, they conducted an experiment using mice as the subjects.

Researchers administered small amounts of THC (4 times less than the intoxicating amount) to mice before simulating a heart attack by restricting their blood flow.
The THC was administered on three different schedules in reference to the “heart attack”: 2 hours prior, 48 hours prior, and 3 weeks prior of continuous treatment.
In order to gauge the effect of THC in this study, researchers observed a number of common signs and/or residual effects of heart attacks. In each schedule of THC administration the study reported an improvement in each of the categories.
THC Has The Upper Hand On Cardiac Damage
Fractional Shortening is a ratio used to objectively rate the level of efficiency that a ventricle is working with. In observing this ratio, researchers found that THC treatment resulted in a 4.7% increase.
Troponin T is a regulatory protein found in cardiac muscle that leaks into the bloodstream in the case of cardiovascular damage. High volumes of it in the blood is often used to diagnose heart attacks. Researchers found that THC treatment reduced the amount of Troponin T by an average of 4 nano grams per milliliter.
The area of dead tissue caused by insufficient blood flow decreased by 6% after THC treatment.
In cases where blood is unable to reach a specific area, an infarction can form. This is an area in which the tissue dies, due to a process called necrosis. The researchers measured the size of these infarctions and found they decreased 6% after THC treatment.
Better heart health undoubtedly results in a longer lifespan. The study above suggests an ultra-low dose of THC can be beneficial in preparation for cardiac surgery. It could limit the damage incurred by the patient and possibly aid in the recovery.
Source: Medical Jane

 

Study: Cannabis Offers Possible Treatment for High Grade Gliomas

What Are High Grade Gliomas?

Gliomas are a type of advanced brain cancer that are fast-growing, aggressive, and difficult to treat. Gliomas occur in the brain and spinal cord and result from the excessive proliferation of abnormal neuroglia, cells of the nervous system that are not directly involved in signaling (as opposed to neurons). Usually, neuroglia work in important, supportive roles to ensure that neurons are communicating properly and efficiently, and there are 3 neuroglia for every 1 neuron in the body. Typesof gliomas include astrocytomas (the most frequent type), ependymomas (the most common glioma in children), and oligodendrogliomas.
There are benign gliomas (which do not invade brain tissue and can be removed if in certain locations, only producing symptoms if they begin to compress important surrounding structures) and malignant gliomas (which do invade brain tissue and have a worse prognosis, or an increased likelihood of shortened survival). So, while not all gliomas are malignant, 80% of malignant brain tumors are gliomas. In the United Kingdom, 1-year survival at diagnosis of a high grade glioma is 36%, and 5-year survival is 10%.
For glioblastoma multiforme, a particularly aggressive high grade glioma, the majority of patients diagnosed pass away within 1 year, and 5-year survival is only 6%. While surgery, radiation, and chemotherapy can be used in attempts to treat high grade gliomas, there are very few to no effective treatment options that increase patient survival, and finding ways to extend the lives of patients is desperately needed.
Cannabinoids as Anticancer Agents
This is not the first time that a study has shown that cannabinoids may be useful as anticancer agents. According to the authors of the study, “Numerous reports highlighting potent activity in vitro [i.e. cells studied outside of the body] and in in vivo [i.e. cells studied inside the body] models have established it as a potential anticancer therapeutic agent in a number of cancer types through processes such as:
induction of apoptosis [i.e. programmed cell death]…
autophagy [i.e. “self-eating”/”self-destruction”] via engagement of the mitogen-activated protein kinase [i.e. an enzyme activated by an agent which induces mitosis, the process by which cells increase in number, which is how cancerous tumors form] and the endoplasmic reticulum [i.e. an organelle, or part of the cell, involved in protein and lipid synthesis] stress-related pathways…
antiangiogeni[sis] [i.e. preventing formation of blood vessels, which cancerous growths need to survive]…
anti-inflammatory
anti-migratory

While the authors note that the psychoactive nature of THC has increased controversy in consideration of cancer treatment with its use,” a pilot trial of its therapeutic use in patients with glioblastoma multiforme (GBM), an advanced type of glioma, showed feasibility without any overt psychoactive effects.”
Additionally, CBD seems to work through similar mechanisms as THC in terms of anti-tumor effects, except it may not exert effects through receptor activation as frequently as THC, and it does not cause psychoactive effects. Further, THC demonstrates analgesic, anti-emetic, and anti-inflammatory properties, whereas CBD possesses anti-psychotic, anti-seizure, and anti-anxiety properties. Together, these cannabinoids (and many more) work together to create an entourage effect that is much more powerful than any single cannabinoid.
Results of the Current Cannabinoid Study
in-Vitro
For the in-vitro portion of the study, the researchers evaluated the effects of THC and CBD on GBM and glioblastoma astrocytoma cells.
Cannabinoids Alone
They found that use of CBD and THC in pure form (>96% purity) and in their whole-plant forms (60% to 72% of each cannabinoid) decreased the number of high grade glioma cells in a dose-dependent manner.
Analysis of the cells showed that there were no changes made to cell DNA, which points to the mechanism of action being one of cytostasis (i.e. prevention of cell growth and proliferation). The effect was greater for GBM cells than for glioblastoma astrocytoma cells.
Rather than causing apoptosis, the cannabinoids seemed to inhibit cell growth and proliferation via autophagy.
Additionally, it was found that CBD was more effective in inhibiting cell growth in its pure form, while THC was more effective in its whole-plant form.
The combination of CBD and THC seemed to have even greater effects (for glioblastoma astrocytoma more than for glioblastoma multiforme), although these results were not statistically significant (i.e. it is fairly likely that this association occurred by chance).
CBD and THC appeared to control cytostasis through mechanisms such as modification of phosphorylation patterns in cell signaling pathways which lead to increased cell growth and proliferation, most significantly through MAPK pathways after use of pure CBD or THC for 4 hours, but also through ERK pathways for glioblastoma multiforme cells (and glioblastoma astrocytoma cells, when CBD was used alone), with potential for modulation through the AKTpathway. At high doses, cannabinoids decreased AKT and and ERK pathway activation, and even more significantly when radiation was used in conjunction.
Cannabinoids Plus Radiation
When cells were pre-treated for 4 hours prior to radiation treatment with pure CBD, pure THC, or a combination of pure CBD and THC, it was found that the combination reduced the number of high grade glioma cells after radiation (i.e. incubation in the combination of cannabinoids made the cells more sensitive to destruction by radiation). Additionally, 5 hours post-radiation, high grade glioma cells that had been treated with cannabinoids had more DNA damage than cells that had been treated with radiation alone (this is a positive result, given that the point of radiation treatment is to damage the DNA of malignant cells in order to cause programmed cell death).
The only time that induction of apoptosis as the antitumor/anticancer mechanism of action was examined was in use of radiation combined with high doses of cannabinoids.
in-Vivo Mouse Model
For the in-vivo portion of the study, the researchers evaluated the effects of THC and CBD on C57BL/6 mice with transplanted GL261 glioma tumors. 80% of mice who received transplants developed tumors, and the researchers found that radiation alone had no effect on glioma growth, while combined THC and CBD inhibited progression, and combined THC, CBD, and radiation inhibited progression the most.
When the brains of the mice were examined after death, it was found that the groups that had been treated with cannabinoids had cancerous growths that were denser than in mice who had not been treated with cannabinoids, suggesting inhibited progression. Additionally, the “vascularization marker” CD31 was reduced in mice who had been treated with cannabinoids (in order to grow and spread, tumors need a blood supply, and increased vascularization is usually a sign of more aggressive growths and a worse prognosis). TUNEL staining was also often increased in the brains of mice who had been treated with cannabinoids, signaling increased DNA fragmentation, especially in combination with radiation treatment (again, this is a positive sign for inhibition of growth/proliferation).
Conclusion
According to the researchers, “…[T]hese data add further support to the concept that cannabinoids both alone and in combination with each other, possess anticancer properties.” Given this exciting new evidence, in conjunction with the previously existing evidence supporting the potential application of cannabinoids as antitumor/anticancer agents, increased research on the use of cannabinoids in treatment of not only high grade gliomas, but also other types of cancerous growths, is warranted and needed as soon as possible.
For information on how you can advocate to move cannabis out of the Schedule I controlled substance classification in order to increase research in the United States, expectations, and safety in considering whole-plant medical cannabis use.
Source : Medical Jane

Hemp offers real sustainable solutions to our earth-killing practices

Hemp, the non-psychoactive strains of the Cannabis family, was once one of the most ubiquitous plants in the world. First found around 8,000 BCE in central Asia, hemp spread across multiple continents through the ages and was a fundamental part of the agricultural revolution. Throughout several civilizations, hemp was used for food, textiles, oil, and industrial purposes. Yet, after getting confused with marijuana in the 1900s, hemp was soon outlawed and forgotten. Many of its benefits were lost in the modern world.

Popular Mechanics published an article back in 1941 with findings that hemp “can be used to produce more than 25,000 products”. In several other countries, hemp has continued to be used for food, textiles, and even in construction to build houses that are more energy efficient than regular buildings.
popular mechanics hemp billion dollar crop

Hemp can help our Farmers and the Planet
The best part of hemp is that its applications are completely eco-friendly and sustainable.

Farmers can actually restore the health of their farmland by planting hemp as it eliminates the need to use agrochemicals such as herbicides or pesticides. Since hemp grows so densely and its roots are so deep, it kills off weeds naturally.

Planting hemp can offer an alternative solution to many of our current practices that are damaging this planet. With a growing cycle of only 4–6 months, hemp is a more sustainable option than trees for paper. Anything you can make out of fossil fuel, you can make out of hemp. This includes energy, plastic, or any other petroleum based products.
hemp’s diverse applications

Hemp has too many applications for us to ignore, especially as we fight an uphill battle against climate change. Bringing hemp back could be key to our sustainability efforts in preserving our soil and natural resources. Developing different applications of hemp and spreading it to the mainstream will help increase the supply of hemp and jumpstart a shift to a healthier society.

It’s time for us to take another look at hemp
We encourage you to learn more about the benefits and uses of hemp by getting involved with hemp events in your city. This is a fantastic way to meet hemp enthusiasts in your community, while helping to grow hemp awareness.

You can also support the hemp movement by writing to your legislators. Ask them to support the Industrial Hemp Farming Act to allow our farmers to grow this crop. Zev Paiss, the founder of the National Hemp Association, claims “The Industrial Hemp Farming Act could be the largest jobs bill that Congress can pass in 2016”.

Source : Ministry of Hemp

Raw chocolate hemp pudding

Ingredients:
1/2 cup water
2/3 cup ice
1 cup hemp seeds
1/8 cup raw cacao powder
1/8 tsp vanilla bean powder
Optional: fresh strawberries, blueberries or raspberries

Method:
Add the water, ice and dates to the blender and blend until you get a consistency of a rough paste.
Add cacao powder, hemp seeds and vanilla bean powder.
Blend everything together until the pudding looks smooth and creamy.
Serve in a bowl by itself or top it up with fresh berries of your choice.
Enjoy

Strawberry Hemp Smoothie

Ingredients:

1 cup organic milk (or milk alternative if you prefer)
1 cup organic vanilla yogurt
1 cup frozen strawberries
1 cup other frozen berries – blackberries, blueberries, raspberries, etc.
1 tsp vanilla extract
¼ cup hemp seeds
2-3 pitted dates for added sweetness (optional)

Method:
Place all ingredients in a blender and pulse until a slush forms.
Blend on high until mixture is smooth and creamy.

Enjoy